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Understanding the interplay between BK polyomavirus and celular restriction factors

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Author
Lovtsov, AlexeyORCiD Profile - 0000-0003-4670-900XWoS Profile - C-2647-2019Scopus Profile - 58235177800
Rjabčenko, BorisORCiD Profile - 0000-0001-7076-256XWoS Profile - U-8964-2017Scopus Profile - 36142246600
Janovec, VáclavORCiD Profile - 0000-0002-6262-3364WoS Profile - N-7608-2017Scopus Profile - 57193086112
Forstová, JitkaORCiD Profile - 0000-0003-0219-506XWoS Profile - L-7328-2017Scopus Profile - 6701385419
Huerfano Meneses, SandraORCiD Profile - 0000-0001-5221-3014WoS Profile - G-8469-2013Scopus Profile - 6506988916

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Publication date
2024
Published in
Czech Chemical Society Symposium Series
Publisher / Publication place
Česká společnost chemická
Volume / Issue
22 (6)
ISBN / ISSN
ISSN: 2336-7202
ISBN / ISSN
eISSN: 2336-7210
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Abstract
The most important human polyomavirus are the Merkel cell polyomavirus, the etiological agent of 80% of Merkel cell carcinoma, JCPyV that causes progressive multifocal leukoencephalopathy and BKPyV which causes nephropathy. After initial primary infection and dissemination in the organism, persistence of polyomavirus is, by not well-known mechanism. Local innate immune response and other cellular restriction factors such as macromolecular protein complexes such as the PML nuclear bodies (PML NB) can play a role in the outcome of the infection and promote viral persistency. Our reseach focus on the understanding the molecular mechanisms of innate immune response activation and modulation in response BKPyV.
Keywords
BKPyV, Merkel cell
Permanent link
https://hdl.handle.net/20.500.14178/2659
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